The vitamin K is in the group of liposoluble vitamins to the like vitamin A; vitamin K is responsible for maintaining hemostasis, that is, coagulation, preventing bleeding from being triggered in the human body.
This can be classified according to its origin in this way: vitamin K1 originating from dark leafy vegetable products, tomato, alfalfa, some cereals and largely found in the liver of pig animals; the vitamin K2 is the product of intestinal bacteria driven processes and finally the vitamin K3 is a variant of artificial origin which is to establish the properties of the aforementioned.
The role of vitamin K in the coagulation process is at the liver level since it is a cofactor of the enzymes in charge of the synthesis of coagulation factors, important proteins to generate the coagulation cascade that is nothing more than a series of events or processes aimed at preventing internal and external bleeding in the patient; In the same way, vitamin K is used for the maintenance of bone metabolism, whose process is in charge of the bone cell called osteocalcin, since it degrades the wrong formations of bone tissue, it is said that vitamin K helps the maturation of this cell indirectly participating in this process, providing density to bone mass and avoiding fractures in people who suffer fromosteoporosis.
The deficiency of vitamin K occurs sporadically and atypical since the only cause is poor intestinal absorption, or can also be presented in long - term treatment with antibiotics, being a patient with symptoms characteristic as bruising and bleeding such as bleeding in the nose (called epitaxis), gum bleeding (gigivorrhagia), abundant menstruation in the case of women and bleeding in the stool (melena), otherwise high levels of vitamin K can occur with hypercoagulability, therefore patients who take anticoagulants have reduced vitamin K intake, however intoxication has never been diagnosedby vitamin K at the time of excessive ingestion of it.
